Narrative Fail In Real Time

Plus A Recommended Read

This morning Robert Malone retweeted nice list explaining vaccine hesitancy in ways that even the slowest vax enthusiasts will hopefully understand. The list, and Malone’s additions, illustrates the type of ongoing narrative fails that lead to distrust—which is an entirely reasonable reaction to flip flopping narratives. Of course, much of this can be traced right back to Tony Fauci who—as Malone put it the other day—every day just says “stuff.”

Robert W Malone, MD @RWMaloneMD

They missed a few things - Herd immunity is the goal. Oops - we will never get to herd immunity Vaccine immunity is stronger natural immunity -oops other way around Unvaccinated spread disease -oops -all spread disease. Escape mutants evading the vaccine - driven by the vaccine

Handwaving Freakoutery @Freakoutery

An explainer to the pro-vax community, explaining the vax-skeptic community. Retweet if you find this helpful. https://t.co/g7R8mNPGqp

Malone also linked to a brief blog post by his wife, Jill Malone: WHAT DO COVID, HIV AND MANY COMMON COLDS HAVE IN COMMON?

The post begins with some technical detail regarding RNA viruses (as opposed to DNA viruses). However, the concluding few paragraphs are eye openers, the implications of which will be very clear.

First, Jill explains that RNA viruses of the type to which coronavirus belongs (there are two basic types) has a very significant capability:

… the RNA genome of a coronavirus …, if transferred into a cell, can cause that cell to produce complete and infectious new coronaviruses. This is why mRNA vaccines only use a fragment of the mRNA genome, so that the mRNA can not reproduce virus.

This capability is related—follow the link for an explanation—to the fact that coronaviruses and, RNA viruses generally, often mutate very rapidly. Which has important implications for would be vaccine developers, as well as for target populations—targets of the virus and targets of vaccine enthusiasts.

Read this slowly:

… viruses that use RNA often mutate very fast. Good thing that human beings use DNA to store their genetic information!

Also a good thing that these mRNA gene therapy medications can’t be assimilated into our DNA—can it?

RNA viruses make this high mutation rate work for them. The high mutation rate of RNA viruses is one reason why it is difficult to make effective vaccines against many of these types of viruses.

This high mutation rate is a two-edged sword. On the one hand, it makes the virus unstable. But there’s a hidden benefit to that—which makes the high mutation rate more of a feature rather than a bug, when it comes to the key ability to evolve and spread:

Positive-sense RNA viruses account for a large fraction of all known human viruses, including many well-known pathogens such as HIV (the AIDS virus), hepatitis C virus (liver cancer), rhinoviruses (common cold), West Nile virus, Dengue virus, Zika, SARS and MERS coronaviruses, and COVID-19. Even though the single stranded RNA strategy comes with the problem of high mutation rate, these viruses replicate so efficiently, and produce so many viruses so fast, that it does not slow them down. In fact, the high mutation rate is sort of an advantage for viruses- it makes it easy for them to evolve and adapt to a new host (you and me) very rapidly, and to adapt to escape immunity in the animals that they infect (including us).

Now, read and reread:

There have been reports of the virus’ genome being different at various time points within an individual. Another RNA virus with this capability that we are all familiar with is HIV.

(For those of you paying attention, smash these ideas together with 1) escape mutants against a vaccine with a target of a single protein (spike in this case) and 2) why we don't have a vaccine for HIV and the common cold...)

Now we see why honest scientists like the Malone’s are stunned and appalled at what’s been going on.